Moisés Rojas Rechy
Structural biology of virus-host interactions Laboratory
Biofisika Institute (CSIC, EHU)
Science Park of the EHU
Barrio Sarriena s/n. 48940 Leioa, Bizkaia (Spain)
Moisés is an early-career researcher with a strong background in structural biology, specializing in the elucidation of molecular mechanisms that govern protein–protein interactions in viral systems. His research focuses on viruses, employing a combination of structural and biochemical approaches to investigate how viral and host proteins assemble, interact, and regulate infection processes.
He earned his PhD from the University of Leeds, where his doctoral work centered on protein–protein interactions and the structural mechanisms underlying herpesvirus biology. This research integrated structural, biochemical, and computational methodologies to explore how viral proteins assemble and interface with host factors to modulate infection and evade immune responses.
Prior to his doctoral studies, Moisés completed a Master’s degree at the Instituto Politécnico Nacional (IPN) in Mexico City, where he investigated herpesvirus-mediated modulation of inflammatory pathways. This work contributed to published studies on the complex interplay between viral infection and host immune responses. His academic journey began with a Bachelor’s degree in Clinical Chemistry from the Universidad Veracruzana, where his interest in molecular virology and host–pathogen interactions first took root.
These diverse experiences have shaped his multidisciplinary approach to studying complex viral systems, integrating virology, structural biology, and computational modeling to uncover molecular insights with biomedical relevance.
His current research interests include the integration of cryo-electron microscopy (cryo-EM), X-ray crystallography, and computational modeling—particularly molecular dynamics-based refinement tools—to achieve high-resolution insights into dynamic protein complexes. His overarching goal is to elucidate the structural principles underlying viral replication and immune evasion, ultimately contributing to the rational design of antiviral strategies and therapeutics targeting critical interaction interfaces.